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Age of Wonders: Shadow Magic for PC was. Age Of Wonders: Shadow Magic for Windows Phone -. The Wizard’s Throne.Q:

DejaVu Sans Mono still rendering with bold, what happened?

I’m using gnome-terminal and the DejaVu Sans Mono font with the console-data package, and I was hoping to see the output bolded:

However I’m getting this instead:

I was wondering what happened in the last 8.04 versions that made it not bold? I’m running Ubuntu 10.04.1 Lucid.

A:

I just realized what happened. The console-data package with DejaVu Sans Mono wasn’t installed.
When I installed it (using sudo apt-get install console-data) then everything worked

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Our recent studies have demonstrated that changes in calcium ion homeostasis are important factors in the pathogenesis of chronic arrhythmias in hearts with coronary artery disease (CAD). These changes could be primarily explained by alterations of calcium ion homeostasis from an imbalance between influx of extracellular calcium into the myocardium and efflux of calcium across the sarcolemmal membranes. Therefore, we investigated whether alteration of cardiac calcium ion exchangers which are the major plasma membrane transporters for calcium ion homeostasis are involved in the pathogenesis of chronic arrhythmias in CAD. Our studies demonstrated that the calcium ion exchangers in the myocardium (NCX, SERCA2, PMCA2 and Na/Ca exchanger) in CAD hearts were significantly decreased compared with those in non-CAD hearts. Furthermore, western blot analysis showed that a decreased expression of SERCA2 and NCX was associated with decreased protein levels of SERCA2 and NCX in CAD hearts. Moreover, the mRNA level of SERCA2 and NCX were also significantly decreased. These studies suggested that the decreased expression of SERCA2 and NCX may be responsible for the alterations of calcium homeostasis in the myocardium of CAD hearts. These ion channel calcium exchangers were restored by corrective procedures such as anti-ischemic drugs or surgical revascularization in experimental CAD animal models. These data suggest that the alteration of ion channel calcium exchangers might be one of the mechanisms
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