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The c-Jun N-terminal kinase (JNK) is known to be activated during HIV-1 infection. In an effort to interfere with this process, we first determined whether JNK inhibitors could be useful for the treatment of the disease. In the second part of the study, the effects of JNK inhibitors on the proliferation and activation of lymphocytes from HIV-1-infected patients were investigated. The JNK inhibitors SP600125 and PAO15928 were found to significantly inhibit the proliferative capacity of lymphocytes, and this was reflected in the frequency of CD3+ and CD4+ cells in the cultures. Moreover, these inhibitors significantly inhibited the activation of T cells, as indicated by a decrease in the expression of activation markers on CD4+ and CD8+ cells. However, they did not have any effect on the phytohemagglutinin-induced proliferative capacity. Our results demonstrate that JNK inhibitors can be useful for the treatment of HIV-1-infected patients.Q:

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